New MBS Items from 1 November 2022

Two new MBS items (11736 and 11737) will be introduced for remote monitoring of cardiac ILR devices. These new services will be remote mirror services of current in rooms service MBS items 11728 and 11731, allowing remote services to be performed.  Refer to the Quick Reference Guide for further details now available on MBS Online.

In addition, 8 eight MBS items for cardiothoracic surgery items 38510, 38513, 38516, 38517, 38555, 38556, 38557 and 38572 will be amended to incentivise the use of advanced techniques and procedures. See full details of these changes on the MBS website.

Claiming Guide for repeat MBS services for stress echo and stress MPS

In response to a number of queries regarding claiming of repeat MBS services for stress echo (item 55143) and stress MPS (items 61349, 61410), the MBS has produced a claiming guide to assist providers.  Link to MBS claiming guide 

Note that there are no changes to the requirements of the services for stress echocardiography and stress MPS, including the qualifying indications, time dependencies or item associations that currently exist.

SURVEY | use of mHealth applications in clinical practice – Cardiovascular Nurses

You are invited to participate in a study being led by the University of Wollongong (UOW) about the use of m-Health applications in clinical practice. The survey is anonymous and takes approximately 10-15 minutes complete.

Wa’ed Shiyab (PhD candidate at UOW) is conducting the survey to examine the use of mHealth applications by nurses for chronic conditions and lifestyle risk factors.

You can access the survey by scanning the QR code or via this link https://redcap.link/mHealthnurse

Study investigators include:  Wa’ed Shiyab (PhD candidate) and Prof Liz Halcomb (Primary Supervisor). Dr Kaye Rolls and A/Prof Caleb Ferguson (Co-Supervisors).

If you have any questions, please contact Mrs Wa’ed Shiyab via email or Prof Liz Halcomb email or phone 4221 3784.

Interviews with 2022 CSANZ ASM Prize Winners

The Education Committee invited the following prize winners to be interviewed after the 2022 CSANZ ASM to talk about their winning work that was presented, and what the future looks like for this project and their work. 

2022 Ralph Reader in Basic and Translational Science Prize Winner
2022 Ralph Reader in Clinical Science Prize Winner
2022 Cardiovascular Nursing Prize Winner
2022 Allied Health, Science & Technology Prize Winner
Dr Dinesh Selvakumar Westmead Institute for Medical Research

Abstract: Cellular Heterogeneity in Human Pluripotent Stem Cell Derived Cardiomyocyte Grafts are responsible for Arrhythmias and are Treatable with Pharmacotherapy and Catheter Ablation.

Read abstract in full here as published in the HLC Journal.

Dr Julia Isbister 
Centenary Institute

Abstract: “Concealed Cardiomyopathy” is an Important Cause of Autopsy-Inconclusive Sudden Cardiac Death and Diagnosis Impacts Care of Surviving Relatives

Read article in full here as published in the HLC Journal.

Kristel Janssens
Baker Heart and Diabetes Institute

Abstract: Modest association between peak exercise blood pressure and ambulatory hypertension in endurance athletes.

Read abstract in full as published in HLC Journal.

Adam Livori
Ballarat Health Services

Abstract: Assessment of Telehealth Cardiology Pharmacist Clinic in Improving Therapy Adherence following Acute Coronary Syndrome and PCI.

  Read abstract in full as published in HLC Journal.

2022 Ralph Reader Prize Winner in Basic and Translational Science Interview

Please find the link below to his winning abstract as published in the Heart Lung and Circulation Journal, August 2022.

2022 Ralph Reader in Basic and Translational Science Prize Winner:
Dr  Dinesh Selvakumar, Westmead Institute for Medical Research.

Cellular Heterogeneity in Human Pluripotent Stem Cell Derived Cardiomyocyte Grafts are responsible for Arrhythmias and are Treatable with Pharmacotherapy and Catheter Ablation

The carbon footprint of hospital diagnostic imaging in Australia

Summary by Dr Scott McAlister and Prof Alexandra Barratt 

As published in The Lancet July 2022.

When we think of carbon (CO2) emissions we don’t often think of healthcare, yet healthcare in Australia is responsible for approximately 7% of national emissions. Approximately 30% of these emissions come from building energy, with most of the rest coming from medical products and processes, such as medical tests, devices and interventions. Carbon footprintingof products and processes involves quantifying emissions from all phases of a product’s life cycle, including extracting raw materials such as ore or gas, manufacturing, use, and then disposal such as through landfill or recycling.

Recent Australian carbon footprinting has been done for pathology testing, intensive care, anaesthesia, and diagnostic imaging. The imaging study showed that one MRI scan has a carbon footprint of 17.5kg CO₂ equivalents (CO2e), which is the same as driving a car 145km, while one CT scan has a footprint of 9.2kg CO₂e, or driving 76km. These are much higher than X-rays (0.76kg CO₂e, 6km) and ultrasound (0.53kg CO₂e, 4km). Reducing imaging or using lower carbon modalities reduces emissions.

One hidden area for healthcare is individuals’ travel for conferences. A return business class flight from Australia to the USA results in approximately 12 tonnes CO2e. By contrast, our individual annual carbon budget in 2030 to keep the world below 1.5°warming will be 2.2 tonnes CO2e.

Authors: McAllister S. et al;  https://doi.org/10.1016/j.lanwpc.2022.100459

ECG of the Month – August 2022

ECG of the Month

A 28 year old nurse with a long history of paroxysmal palpitations has the following 12-lead ECG and 2-channel telemetry trace recorded during an episode.

Which of the following is the diagnosis?

  1. Orthodromic AV reentry tachycardia
  2. AV nodal reentry tachycardia
  3. Focal atrial tachycardia
  4. Slow atrial flutter
  5. Inappropriate sinus tachycardia

 

Explanation:

Panel A displays a short RP narrow complex tachycardia at a rate of approximately 160bpm. This is a supraventricular tachycardia (SVT). P waves can best be appreciated in the inferior limb leads, aVR and V1. There is a 1:1 RP relationship with the P wave falling in the first half of the RR cycle (short RP).

The P wave configuration is deeply negative in inferior limb leads and positive in aVR. It is isoelectric in the first half of V1 before becoming positive in the second half. This morphology is characteristic of a posteroseptal origin of atrial activation but is not diagnostic of tachycardia mechanism. It does however exclude option e) inappropriate sinus tachycardia, because this P wave configuration is inconsistent with a sinus origin of activation.

Panel B shows the rate of SVT has slowed to approximately 140bpm. All 12 leads are no longer available for P wave morphology analysis but the P wave appears unchanged in lead II. Notice that the RP interval is unchanged despite the slower tachycardia rate. Towards the end of the panel, we see a cycle where there is a dropped P wave (purple circle in between red arrows highlighting P waves). This phenomenon is seen again further along in the bottom of the panel. Tachycardia continues without reset despite the dropped P wave. This implies that atrial depolarization is not an obligate component of the tachycardia mechanism and thus option a) orthodromic AV reentry tachycardia can be excluded. This is because orthodromic AV reentry is due to the presence of a large loop circuit comprised of AV node, His-Purkinje system, ventricular myocardium, retrograde conduction to the atrium via an AV bypass tract, atrial myocardium, and then finally, the AV node again. Tachycardia is interrupted if any of these structures fail to depolarize. The dropped P waves seen here exclude orthodromic reentry because tachycardia continues at the same rate and is unaffected by the lack of atrial depolarization.

Additionally, it is readily apparent that with the dropped P waves the overall atrial rate is now lower than the ventricular rate. Consequently, the atrium cannot be driving this narrow complex tachycardia, hence options c) focal atrial tachycardia, and d) slow atrial flutter, can be excluded.

This leaves the correct answer as b) AV nodal reentrant tachycardia (AVNRT).

AVNRT is the most common form of SVT and is caused by reentrant excitation around perinodal transitional zone tissue. Neither atrial nor ventricular myocardium is required for ongoing AVNRT. While conduction block to the ventricle is commonly seen during AVNRT, block to the atrium is rarer. When conduction block to the atrium is seen during a narrow complex tachycardia however, it is very useful as it excludes most other common SVT mechanisms.

Provided by A/Prof Haris M. Haqqani – ECG of the Month – August 2022

 

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